Childhood: Clinical and School Psychology – Lecture 3 (UNIVERSITY OF AMSTERDAM)

About 5% of the children and 2.5% of the adults are diagnosed with ADHD. There are several requirements for a diagnosis:

  1. There are at least six symptoms for at least six months. For people older than 17 years old, there need to be five or more symptoms.
  2. The symptoms are present before the 12th year of age.
  3. The symptoms exist in two or more areas (e.g. school; home).
  4. The symptoms interfere with social, school or professional functioning.
  5. The symptoms are not better explained by other disorders.

There are three subtypes:

  • Predominantly inattentive subtype (i.e. ADD).

    • Careless attention to detail.
    • Problems sustaining attention over time.
    • A poor follow-through (e.g. schoolwork; homework; chores).
    • A poor ability to sustain mental attention (e.g. independent work at school).
    • The child loses necessary materials (e.g. assignment sheets).
    • The child is forgetful.
    • The child is easily distracted.
    • The child is poorly organized.
    • The child does not appear to listen.
  • Predominantly hyperactive/impulsive subtype
    • The child demonstrates fidgety or squirmy behaviour.
    • The child has problems remaining seated.
    • The child shows excessive motion.
    • The child has problems engaging in quiet play.
    • The child is constantly on the go.
    • The child shows incessant talking.
    • The child blurts out answers or comments (i.e. impulsivity).
    • The child is impatient and has problems with turn-taking (i.e. impulsivity).
    • The child is intrusive to others (i.e. impulsivity).
  • Combined subtype.

Children with the inattentive presentation are often misdiagnosed and misunderstood. It is often misinterpreted as a lack of motivation. The children may be characterized by a sluggish information processing style and there are problems with focused or selective attention. These children are unable to filter essential from non-essential details. This lack of attention may result from information overload and the inability to selectively limit the focus of attention. They have poor performance on tests because of this problem with attention and their poor concept of time and time management.

Children with the hyperactive-impulsive presentation experience academic problems due to their impulsive nature. They emphasize speed over accuracy and approach tasks incorrectly because they do not wait for all instructions. They are at social risk due to their impatient nature and they often have poor social skills and difficulties making and maintaining friends. They often gravitate towards other rule-breaking children.

A difficult temperament (1), poor sleep patterns (2), excessive activity (3), irritability (4) and a difficulty soothing the child when they are upset (5) are risk factors in infancy for the development of ADHD. During toddlerhood, the child may demonstrate higher levels of underregulated behaviours and a lack of self-control may persist during the transition to preschool.

The symptom presentation of ADHD differs with age. Inattention symptoms are consistent over time but the hyperactive/impulsive symptoms decrease over time. There is a different ADHD symptom presentation by gender. Females typically have more inattentive symptoms and more internalizing problems while males have more hyperactive/impulsive symptoms. ADHD is more prevalent in males than in females but this may be due to the hyperactivity/impulsivity symptoms being more noticeable than the inattentive symptoms.

There are four biological features of ADHD:

        1. Brain structure
          There is less activity in the frontal brain regions (i.e. executive functioning) and more activity in the cingulate gyrus (i.e. focusing of attention).
        2. Genetic transmission
          Half of the children with ADHD have a parent with ADHD and 75% of the aetiology may be contributed to genetic factors.
        3. Neurotransmitters
          There may be low catecholamines (e.g. dopamine; epinephrine; norepinephrine) in children with ADHD and this is associated with attention and motor activity.
        4. Neurocognitive processing
          There may be a dysfunction in executive functioning.

The altered perception of time in ADHD is associated with reductions of dopamine in the basal ganglia. It may also be the result of dysfunction in the parietal lobe. Medication for ADHD (e.g. Ritalin) increases the number of catecholamines in the brain.

ADHD is very comorbid with other disorders (e.g. ODD). At least 62% has a co-occurring disorder and 34% has at least two co-occurring disorders. Symptoms of bipolar disorder and depression may be mistaken for ADHD. Children with bipolar disorder demonstrate rapid shifts of mood compared to lengthy moods for adults, making differential diagnosis very difficult.

For ADHD, different problems occur at different ages:

  • Preschool
    The most prevalent problems here are behavioural problems and hyperactivity.
  • School-age
    The most prevalent problems here are academic problems (1), social problems (2), low self-esteem (3), oppositional behaviour (4) and accidents (5).
  • Adolescence
    The most prevalent problems here are planning problems (1), social problems (2), low self-esteem (3), addiction (4), behavioural problems (5) and antisocial behaviour (6).
  • Student
    The most prevalent problems here are cognitive under-functioning (1), difficulty finding a job (2), performance anxiety (3), substance abuse (4) and accidents (5).
  • Adulthood
    The most prevalent problems here are job-related problems (1), low self-esteem (2), substance abuse (3), accidents (4) and relation problems (5).

People with ADHD get in more accidents due to risk-taking behaviour. Children who receive medication for ADHD are less prone to substance abuse because they develop resilience and they do not self-medicate.

Barkley’s model of ADHD states that a child’s degree of success in behavioural inhibition is central to determining the outcome of working memory (1), self-regulation (2), internalization of speech (3) and reconstitution (4). Deficits in behavioural inhibition result in poor problem-solving strategies based on an inability to integrate and coordinate information generated by the four central processes. This model may explain the hyperattentive-impulsive presentation of ADHD.

According to this model, children with the hyperattentive-impulsive presentation have problems with sustained information and not with selective attention. Sustained attention consists of attention for effortful tasks and contingency-based attention (i.e. self-rewarding attention). Children have fewer problems with contingency-based attention in novel situations giving the false idea that these children are focused when they want to.

The dual pathway model of ADHD states that ADHD is explained by impairment in executive functioning and the motivational and reward system. Executive functions enable goal-oriented behaviour. Motivation does not refer to deliberate motivation.

Executive function is a control process of the brain and includes attention (1), inhibition (2), working memory (3), flexibility (4), planning (5), problem-solving (6), reasoning (7), initiation of action (8) and monitoring of action (9).

A child with executive functioning dysfunction is typically not annoyed by their own behaviour whereas it is frustrating for the teacher. There are several guidelines for helping a child with ADHD in the classroom with executive functioning problems:

  • Do not get annoyed.
  • Attempt to avoid distractions.
  • Make sure that the task is feasible
    • Provide structure.
    • Give learning objectives (i.e. primary and secondary).
    • Give clear instructions.
    • Model tasks or indicate steps.
    • Repeat important information.
    • Control whether the child can make independent work.

ADHD is highly prevalent and has a different symptom presentation for everybody. The consequences of the disorder can be great and children with ADHD often show executive functioning problems. This, in turn, often causes academic problems. Parents and teachers can help alleviate some of the problems.

Children with ADHD often have deficits in motivation to perform due to dopaminergic system abnormalities. This means that they have (more) motivation to perform self-rewarding tasks with direct rewards (e.g. videogames) but not for other tasks. Children with these deficits need direct rewards (1), more rewards (2) and higher rewards (3). They take longer to learn new behaviour.

Typically developing children already have optimal performance with feedback only. However, the performance of children with ADHD improves with reward and there is more improvement with higher rewards but the performance of children with ADHD does not normalize, regardless of the reward. Children with ADHD have a decrease in performance over time but the performance over time normalizes when given a high reward. This implies that children with ADHD need more motivation, especially on long tasks.

There are several guidelines for helping a child with ADHD in the classroom with motivational problems:

  • Do not get annoyed.
  • Make sure that the task is feasible.
  • Make sure that there is strong motivation.
    • Give direct feedback.
    • Be consistent in giving feedback.
    • Reward more often for longer tasks.
    • Find out what is rewarding for the child.
    • Indicate when movement is permitted.

Every school transition (e.g. other school; other grade) is a challenge for children with ADHD. However, parents and teachers can help by offering feasible tasks and rewards.

There may be a socially disabled subtype for children with ADHD symptoms and a discrepancy between social skills and cognitive ability. Oppositional defiant disorder (ODD) is very common in children with ADHD. It consists of a pattern of negative, hostile and rebellious behaviour. The prevalence of this is 3.3% but it often co-occurs with ADHD. 50% of the children with the combined subtype of ADHD have ODD and 25% of the inattentive type have ODD. There are several requirements for a diagnosis:

  1. For at least six months, four or more symptoms of angry or irritable mood (1), argumentative or defiant behaviour (2) or vindictiveness (3).
  2. The symptoms cause distress to self or others or it impacts social, school or professional functioning.
  3. The symptoms do not exclusively occur during the course of a psychotic, substance use, depressive, bipolar or disruptive mood dysregulation disorder.

Common behaviour in ODD is a bad mood (1), arguing with adults (2), not obeying rules (3), aggression (4), destructive temper tantrums (5), irritating others on purpose (6), blaming others (7), being quickly irritated by others (8) and being vengeful (9). For children younger than five years, this behaviour needs to occur on most days. For children older than five years this behaviour needs to occur at least once a week.

What is normal naughty behaviour changes over the course of development. For example, temper tantrums are common in toddlerhood and naughty behaviour is common in preschool when the child’s goals are blocked (e.g. getting the ball). Problem behaviour is often seen in preschool years but is often unlearned over time.

Aggression in the preschool years is an important predictor of aggression in later life. There are three pathways to problem behaviour and delinquency:

  1. Authority conflict pathway
    This starts with stubborn behaviour which starts before the age of 12. This develops into defiance and disobedience and then into authority avoidance (e.g. truancy). This can merge with the other pathways.
  2. Overt pathway
    This starts with minor aggression (e.g. bullying; annoying others) and develops into physical fighting and, lastly, violence.
  3. Covert pathway
    This starts with minor covert behaviour (e.g. shoplifting) and develops into property damage and, lastly, serious delinquency (e.g. fraud; burglary).

The different pathways can overlap as it develops. There are several predictors of problem behaviour and these predictors are hereditary, rather than aggression itself.

  • Problem behaviour at a young age.
  • The male sex.
  • Attention problems.
  • Hyperactivity.
  • A difficult temperament.
  • A low resting heart rate.
  • A low serotonin level.
  • A low IQ.
  • Limited pro-social emotions.

The environment plays an important role in the development of aggression. There are several family characteristics in the case of child aggression:

  • Inconsistent application of rules.
  • Physical and harsh punishment.
  • Lack of warmth, sensitivity and monitoring.
  • A low SES, unsafe environment and little social network.

There is often an attraction to peers with similar behaviour. If a child shows problem behaviour, this leads to many short-term relationships and many conflicts.

The learning theory states that problem behaviour is learned as a result of wrong instructions (1), wrong examples (2), endorsement of wrong behaviour (3) and through coercive interactions (4). Overlearned activities require very little active cognitive processing which makes it possible to do something else at the same time (e.g. driving and talking).

Coercive interactions consist of interactions where behaviour is enforced on one another and little attention is paid to prosocial behaviour. A coercive cycle can be parent-driven or child-driven. Coercive sequences are not pathological by themselves. A lot of the aversive events (e.g. attack) are positively reinforced by the victim (e.g. give up bicycle; cry), leading to an increase in the probability of future aversive events on the victim. Peers and siblings may be an important source of positive reinforcement, especially for physical aggression (e.g. hitting).

Coercion may be overlearned. The effects of reinforcement and punishment contingencies in family interaction sequences may be automatic. This means that this interaction is not mediated by thought or expectancies. Family therapy is very difficult because the coercion process is automatic.

 It may be useful to bring the overlearned aspects of the coercion process in the open. This can be done by This can be done by providing clearly stated directives in a neutral affect and follow-up with positive reinforcement for cooperation and small negative sanctions for non-compliance.

The contingency theory states that everybody changes their behaviour to match the changes in contingencies one experiences in their social environment. The contingencies may operate outside of family members’ awareness or intention, meaning that they may result in increasing reliance on aversive behaviour as a means of influence and control. This may lead to extended conflict which escalates in intensity.

Negative reciprocity is more likely when one member of a family engages in coercive behaviour (e.g. when the mother engages in aversive behaviour, the child is more likely to also respond negatively). This leads to a sequence that increases coercion:

  1. Frequent aversive behaviour sets up reinforcement contingencies.
  2. The contingencies are followed by increases in chains of aversive behaviour (i.e. repeated aversive behaviours by multiple family members).
  3. The aversive behaviours are accompanied by outbursts of negative emotion.

The presence of negative affect increases the risk and longer chains of aversive events are more likely to escalate in intensity. The negative reinforcement contingency that accompanied an aversive exchange is more powerful and resistant to extinction than positive reinforcement. This may shape aversive behaviour.

Infants may use negative contingencies to teach caregiving skills to family members (e.g. crying as an aversive behaviour). The differences in reactions of both the infant and the parents influence the risk for persistent involvement in coercive social processes (e.g. time spent crying before the caregiver picks up the infant). In most families, teaching the child to use words and positive behaviour gradually replaced coercion but it can persist to early childhood. Coercive behaviour peaks around the age of 3 (e.g. temper tantrums of toddlers).

The degree to which child coercive behaviours persist depends on the effectiveness of parents’ use of contingencies. The preschool period can be a period of growth in coercion, depending on parental skills and child temperament. There are three stages in the sequence of coercive behaviours:

        1. Stage 1: the family (i.e. infancy and toddlerhood)
          There can be coercive interactions between the infant and the mother. As the child ages, more family members are involved and it increases in intensity. At this point, several coercive processes may begin to run simultaneously. There is a progression to a wider variety of coercive behaviours shaped by reinforcement contingencies during family interactions. This stage includes negative reinforcement for coercive behaviours.
        2. Stage 2: the deviant peer group (i.e. school age through adolescence)
          The reinforcement contingencies are positive (1), are provided by peers (2) and shape more covert forms of aggression (3). Friends’ reinforcement accounts for a lot of variance of adolescent rule-breaking or deviant talk (i.e. deviancy training by peers). The contribution of deviant peers could begin as early as school entry.
        3. Stage 3: peer deviancy training
          Deviant peers shape increasingly deviant behaviour through both negative and positive reinforcement. There are significant contributions to the coercion process by both the family and the coercive peer group.

Children who started aggressive and oppositional behaviour early in development were more at risk for police arrests and to become chronic offenders than late starters. Risk for police arrest increases as children move through the stages of coercion.

The macro-level of coercion describes the reactions people have when confronted by some form of antisocial behaviour (e.g. rejection of peer that shows antisocial behaviour). It is also associated with changes in the form of deviant behaviour that evolve from people’s reactions to antisocial behaviour.

As children move to stage three, they come into contact with a subgroup of peers who are even more extreme. They will combine elements of stage 1 with those of stage 2. There is a drop in positive parenting and monitoring as the family becomes increasingly coercive. These children drift into increasingly deviant environments through reinforcement.

The Parent Management Training-Oregon model (PMTO) is suitable for treatment and prevention of antisocial behaviour. It leads to more effective parenting (1), less child externalizing problems (2), less maternal depression (3), higher SES (4) and lower deviant peer association (5). The work-struggle hypothesis refers to the relationship between therapists and clients where the therapist suggests change and the client resists it.

The social interaction learning model (SIL) states that ongoing coercive behaviour is governed by its positive and negative reinforcement contingencies and not cognitive processes. This seems to be supported. A reduction in coercion is typically associated with a growth in positive parenting. Coercion and a lack of positive parenting contribute to growth in deviancy.

The information processing theories differentiate between reactive aggression and proactive aggression. According to these theories, reactive aggression is caused by rejection experiences. There is an aggressive experience (1), this leads to selective attention to threatening information (2), this leads to a negative interpretation of other people’s behaviour (3), this leads to anger (4), this leads to a difficulty in regulating the anger (5) which, in turn, again leads to an aggressive experience.

Proactive aggression is caused by observational learning and reinforcement of aggressive behaviour. The child has dominance and revenge goals (1), limited social skills (2) and an overestimation of approval of others on aggressive behaviour (3) and this leads to proactive aggression.

Aggression always has a reason. It is typically an expression of dissatisfaction or distress and can be the result of depression (1), anxiety (2), overestimation of situation (3), cognitive dissonance (4) or relationships with others (5). It is a coping style.

Psychoeducation can help families to communicate better. It consists of learning how to bear the feeling (1), communicate about emotions (2), learning to talk about difficult subjects when calm (3) and understanding the roots of the anger (4).

For the assessment of ADHD, a semi-structured interview is useful. It is important to use multiple informants (1), obtain information concerning other areas of diagnostic concern (e.g. depression) (2) and it may be useful to have a cognitive assessment to evaluate deficits in specific areas (3).

Treatment alternatives for ADHD depend on the comorbid features (1), symptoms (2) and the nature and extent of functional impairment (3). The interventions can be applied at home (1), at school (2) or in interactions with peers (3). Stimulant medication is more effective than behavioural therapy in alleviating core symptoms of the disorder but there are no long-term differences.

Anxious children with ADHD may benefit equally well from behaviour therapy compared to medication. Parent training can improve parenting skills (1), reduce parental stress (2), reduce core symptoms of ADHD (3) and reduce non-compliance (4).

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Childhood: Clinical and School Psychology – Lecture summary (UNIVERSITY OF AMSTERDAM)

Childhood: Clinical and School Psychology – Lecture 1 (UNIVERSITY OF AMSTERDAM)

Childhood: Clinical and School Psychology – Lecture 1 (UNIVERSITY OF AMSTERDAM)

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A case formulation refers to a hypothesis about why the problem behaviour exists and how it is maintained. This should be based on the longevity of the problems (1), consistency of problematic behaviour across situational contexts (2) and family history (3). Problematic behaviour is characterized by the four d’s:

  1. Deviance from the norm
    This refers to determining the degree to which behaviours are deviant from the norm. This can be assessed using informal testing (e.g. interview) or formal tests (e.g. test batteries).
  2. Distress
    This refers to assessing the relative impact of a disorder after the disorder has been identified.
  3. Dysfunctional
    This refers to assessing the distress that a disorder causes. For children, multiple sources of information may need to be used.
  4. Dangerous
    This refers to assessing whether there is a risk for self-harm and a risk of harm to others.

The duration should also be taken into account. Clinical decisions are often taken based on measures of intensity (1), duration (2) and frequency of the behaviour relative to the norm. To have a valid diagnosis, several things need to be taken into account:

  • It is important to have knowledge of normal behaviour and deviant behaviour during development.
  • It is important to take equifinality (i.e. different factors having the same outcome) into account.
  • It is important to take multifinality (i.e. the same factor leading to different outcomes) into account.
  • It is important to make use of a multimethod approach (i.e. a variety of informants and procedures).

It is essential to take the developmental stage of a child into account when assessing behaviour. According to Erikson, children develop through psychosocial stages with socioemotional tasks that must be mastered to allow for positive growth across the lifespan (e.g. trust vs. mistrust). Behavioural theories state that behaviour is shaped by associations (i.e. contingencies) resulting from positive and negative reinforcement.

There are three questions that need to be answered by the clinician after assessment:

  1. “What are the characteristics of the child’s problem?”
  2. “How should the problem be evaluated?”
  3. What are appropriate intervention strategies?”

The adaptation theory states that early attachment relationships have an impact throughout the lifespan. Triadic reciprocity refers to the dynamic system between the person, the environment and behaviour where all three influence each other.

Family systems theory states that the family is a system made up of subsystems (e.g. parent and child; parent and parent). The behaviours in a system are aimed at maintaining or changing boundaries, alignment and power. A family’s degree of dysfunction can be determined by boundaries that are poorly or inconsistently defined.

The DSM is a categorical diagnostic system. It makes use of clear-cut categories as this is needed for health care service (e.g. insurance). However, there are several problems with this:

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Childhood: Clinical and School Psychology – Lecture 2 (UNIVERSITY OF AMSTERDAM)

Childhood: Clinical and School Psychology – Lecture 2 (UNIVERSITY OF AMSTERDAM)

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Fear refers to the emotional response to real or perceived immediate threat. Anxiety refers to the anticipation of future threat. Phobia refers to being fearful or anxious about or avoidant of a certain object or situation. There is no specific cognitive ideation.

There are four symptoms of anxiety:

  1. Emotional symptoms
    This includes an anxious feeling.
  2. Cognitive symptoms
    This includes negative thoughts and a tunnel vision.
  3. Physiological symptoms
    This includes trembling, palpitations, sweaty hands, tension, headache and abdominal pain.
  4. Behavioural symptoms
    This includes avoidance.

The purpose of anxiety is to alarm one of danger (1), prepare the body to act quickly (2) and keep distance from the danger (3). There is no strict boundary between typical and deviant anxiety. Therefore, the four d’s need to be evaluated when assessing anxiety.

What a child is afraid of changes with age. Children may be more afraid of animals due to their magical thinking. They may be afraid of the dark because of their inability to control it. At seven or eight years old children start to become more concerned with the future.

There is an increase in the prevalence of any anxiety disorders from the age of 11. There is a strong decline in separation anxiety disorder after the age of 10. For phobias, the prevalence can reach up to 16% in adolescence.

Children who suffer from anxiety disorders will be clinging (1), show physical complaints (2), have sleep problems (3), concentration problems (4), avoid certain situations (5), demonstrate a lot of ‘just in case’ behaviour (6), feel small (7), get angry (8), get easily upset (9), demonstrate perfectionism (10), stay home from school (11), lie (12) and alarm adults (13). There is comorbidity between ADHD and anxiety disorders.

Behavioural inhibition refers to inhibited behaviour. As an infant, there is high reactivity to stimuli (e.g. crying). In preschool, these children do not approach strangers (1), stare at strangers (2) and stays close to the mother (3). Modelling behaviour is stronger in the same-sex parent.

There are several risk factors for anxiety disorders:

  1. Genotype of the child on phenotype of child effects
    This includes temperament (e.g. behavioural inhibition) (1), fear sensitivity (2) and disgust sensitivity (3).
  2. Genotype of the parent on rearing environment of child effects
    This includes modelling of anxious behaviours by the parents. This facilitates negative, threatening interpretations of ambiguous situations.
  3. Genotype of the child on rearing environment of child effects
    The temperament of the child influences the degree of control and protection from emotionally upsetting situations by the parents.
  4. Phenotype of the child effects
    This includes avoidance (1), negative thoughts (2), feeling sick (3) and feeling anxious (4).
  5. Rearing environment of child on phenotype of child effects
    The rearing environment of the child can stimulate and
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Childhood: Clinical and School Psychology – Lecture 3 (UNIVERSITY OF AMSTERDAM)

Childhood: Clinical and School Psychology – Lecture 3 (UNIVERSITY OF AMSTERDAM)

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About 5% of the children and 2.5% of the adults are diagnosed with ADHD. There are several requirements for a diagnosis:

  1. There are at least six symptoms for at least six months. For people older than 17 years old, there need to be five or more symptoms.
  2. The symptoms are present before the 12th year of age.
  3. The symptoms exist in two or more areas (e.g. school; home).
  4. The symptoms interfere with social, school or professional functioning.
  5. The symptoms are not better explained by other disorders.

There are three subtypes:

  • Predominantly inattentive subtype (i.e. ADD).
    • Careless attention to detail.
    • Problems sustaining attention over time.
    • A poor follow-through (e.g. schoolwork; homework; chores).
    • A poor ability to sustain mental attention (e.g. independent work at school).
    • The child loses necessary materials (e.g. assignment sheets).
    • The child is forgetful.
    • The child is easily distracted.
    • The child is poorly organized.
    • The child does not appear to listen.
  • Predominantly hyperactive/impulsive subtype
    • The child demonstrates fidgety or squirmy behaviour.
    • The child has problems remaining seated.
    • The child shows excessive motion.
    • The child has problems engaging in quiet play.
    • The child is constantly on the go.
    • The child shows incessant talking.
    • The child blurts out answers or comments (i.e. impulsivity).
    • The child is impatient and has problems with turn-taking (i.e. impulsivity).
    • The child is intrusive to others (i.e. impulsivity).
  • Combined subtype.

Children with the inattentive presentation are often misdiagnosed and misunderstood. It is often misinterpreted as a lack of motivation. The children may be characterized by a sluggish information processing style and there are problems with focused or selective attention. These children are unable to filter essential from non-essential details. This lack of attention may result from information overload and the inability to selectively limit the focus of attention. They have poor performance on tests because of this problem with attention and their poor concept of time and time management.

Children with the hyperactive-impulsive presentation experience academic problems due to their impulsive nature. They emphasize speed over accuracy and approach tasks incorrectly because they do not wait for all instructions. They are at social risk due to their impatient nature and they often have poor social skills and difficulties making and maintaining friends. They often gravitate towards other rule-breaking children.

A difficult temperament (1), poor sleep patterns (2), excessive activity (3), irritability (4) and a difficulty soothing the child when they are upset (5) are risk factors in infancy for the development of ADHD. During toddlerhood, the child may demonstrate higher levels of underregulated behaviours and a lack of self-control may persist during the transition to preschool.

The symptom presentation of ADHD differs with age. Inattention symptoms are consistent over time but the hyperactive/impulsive symptoms decrease over time. There is

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Childhood: Clinical and School Psychology – Lecture 4 (UNIVERSITY OF AMSTERDAM)

Childhood: Clinical and School Psychology – Lecture 4 (UNIVERSITY OF AMSTERDAM)

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Autism spectrum disorder (ASD) is a neurodevelopmental disorder with five criteria:

  1. Persistent deficits in social communication or social contact across multiple contexts
    1. Deficits in social-emotional reciprocity (i.e. visible in toddlers)
      1. Strange way of making contact.
      2. Reduced sharing of interests, pleasure, emotions or affect.
      3. Inability to initiate and respond to interactions.
      4. The child is untuned (e.g. ignoring the listener).
    2. Deficits in non-verbal communicative behaviours used for social interaction (i.e. visible in toddlers)
      1. Limited eye contact.
      2. Odd body language (e.g. turning away).
      3. Limited facial expressions and understanding.
      4. Few gestures and limited understanding of gestures.
    3. Deficits in developing, maintaining and understanding relationships.
      1. Not a lot of fantasy play.
      2. Difficulties in making friends.
      3. Limited empathy.
      4. Difficulty playing together.
      5. Not a lot of interest in peers.
  2. Restricted, repetitive patterns of behaviour, interests or activities (at least two)
    1. Stereotyped or repetitive motor movements, use of objects or speech.
    2. Insistence on sameness, inflexible adherence to routines or ritualized patterns or verbal or non-verbal behaviour.
    3. Highly restricted, fixated interests that are abnormal in intensity or focus.
    4. Hyper- or hypo-reactivity to sensory input or unusual interest in sensory aspects of the environment.
  3. Present in early development (i.e. around two years of age)
  4. Distress and reduced functioning
  5. The symptoms are not better explained by another disorder

Symptoms of autism may not manifest themselves in early development if there is a mild version of the disorder. Most children with ASD will demonstrate difficulties in the key symptom clusters but the symptomatology and severity differ. The severity is based on support needed. This ranges from support to very substantial support.

In ASD, there is abnormal language development and several language difficulties are associated with ASD. The child with ASD:

  • Does not babble.
  • Shows delayed language development (e.g. no use of functional words at 18 months).
  • Shows inappropriate use of language (e.g. difficult words at early age).
  • Makes use of abnormal language (e.g. repeating words; monotonous; long-winded).
  • Takes language literally.
  • Mostly talks about the favourite subject.

There are several symptoms of Asperger’s syndrome:

  • Social impairment and restricted behaviour.
  • There is no speech delay and no cognitive delay.
  • There is specialized knowledge in restricted domains.
  • There are eccentricities (e.g. formal speech; stiff speech).
  • There are difficulties in comprehending non-literal use of language.

While many people with Asperger’s syndrome see it as part of their identity, it was removed from the DSM-5 together with PDD-NOS (1), childhood disintegrative disorder (2) and autistic disorder (3). Asperger’s may not be severe enough to classify as part of the autistic spectrum. However, Asperger’s syndrome may be a separate category due to differences in language development.

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Childhood: Clinical and School Psychology – Lecture 5 (UNIVERSITY OF AMSTERDAM)

Childhood: Clinical and School Psychology – Lecture 5 (UNIVERSITY OF AMSTERDAM)

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The WISC-IQ test measures the following:

  • It measures how well somebody can learn.
  • It measures what somebody knows.
  • It measures what somebody can do.
  • It assesses what a person’s strong skills are.
  • It assesses what a person’s weak skills are.

The focus of an IQ test is on school-based skills (e.g. language; math; understanding; spatial skills; planning; problem-solving; logical skills) and it also measures acquired knowledge and skills. This means that an IQ test does not only measure potential but also current intelligence. It is important to assess whether deficits are the results of lack of opportunities (i.e. deprivation) or limitations in capacity.

There is not a perfect relationship between school performance and IQ as school performance may be lower than somebody’s IQ due to socio-emotional circumstances (e.g. being bullied) or other issues making school performance more problematic (e.g. dyslexia).

An IQ score is not a school advice in the Netherlands. The schools give an advice and CITO scores and IQ scores could change this advice. The traditional view of giftedness holds that a child with an IQ of 130 is gifted. The current view of giftedness holds that there needs to be high ability but not necessarily an IQ score of 130 or higher. Somebody is not only gifted if somebody has potential but also if this person shows gifted behaviour.

According to Renzulli, giftedness does not depend on a single criterion (e.g. IQ) but refers to the interaction between three clusters of traits;

  1. Above-average general abilities (e.g. IQ).
  2. High levels of task commitment (e.g. perseverance).
  3. High levels of creativity.

This definition is applicable to any valuable area of performance (e.g. music; arts) and not just academics. People who are gifted score above average on each of the clusters but not necessarily in the superior range. Whether somebody is gifted depends on the needs and values of a culture.

Persistence in the accomplishment of ends (1), integration toward goals (2), self-confidence (3) and freedom from inferiority feelings (4) are personality factors that predict achievement among individuals with high intellect.

While intelligence on IQ test is relatively stable it does not say anything about development. Giftedness focuses on exceptional behaviour. Gifted children need educational opportunities that are normally not provided in regular instruction. However, this may be needed for all children but the outcomes differ depending on talent.

According to Gardner, there are multiple intelligences. This includes the following:

  • Linguistic intelligence.
  • Logical-mathematical intelligence.
  • Spatial intelligence.
  • Bodily-kinaesthetic intelligence.
  • Musical intelligence.
  • Interpersonal intelligence.
  • Intrapersonal intelligence.
  • Naturalist intelligence.

According to this view, giftedness includes a biopsychological potential to process information. This can be activated by the appropriate cultural setting (e.g. teachers should present lessons through a variety of methods, such as music).

Sternberg’s

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Childhood: Clinical and School Psychology – Lecture 6 (UNIVERSITY OF AMSTERDAM)

Childhood: Clinical and School Psychology – Lecture 6 (UNIVERSITY OF AMSTERDAM)

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Multilingualism refers to speaking multiple languages. It is not just a technical task which only requires a cognitive dimension as it also has emotional, social and cultural significance. There are three dimensions for describing people’s associations with the languages they speak:

  • Expertise
    This refers to the degree of proficiency in a language.
  • Affiliation
    This refers to the affective relationship with a language.
  • Inheritance
    This refers to the membership of a certain language group (e.g. by birth; family; community).

Simultaneous multilingualism (i.e. balanced bilingual) refers to learning the first and second language simultaneously. Both languages are learned in the home environment and the level of both languages is maximal depending on socioeconomic status and input. Successive multilingualism (i.e. functional bilingual) refers to learning the first language first and the second language after mastering the first one. The first language is learned in the home environment whereas the second language is learned at school or work. The level of the first language is maximal but the level of the second language varies. This multilingualism can be domain-specific (e.g. only reading; only speaking).

Lower order processes refer to the level of letter and word recognition. Higher-order processes refer to the comprehension of the content of text. Efficient lower-order processes allow to allocate optimal attention to the interpretation of meaning communicated in the text. Metacognitive skills in reading refer to the ability to use strategies to regulate the reading process.

When learning a language at home, exposure to the best language is essential. It appears as if the first language skill can predict second language learning early on in life. This could help with early diagnoses of reading and spelling problems of the second language. There are several theories as to how the first language influences learning the second:

  1. Linguistic interdependence hypothesis
    This states that there is knowledge transfer from the first to the second language. There is metalinguistic transfer and higher-order skills (e.g. reading strategies) can be transferred from the first to second language. This is believed to aid language acquisition.
  2. Threshold hypothesis
    This states that there needs to be a knowledge threshold before higher-order skills can be transferred. This holds that reading strategies from the first language can be used in the second language when there is enough knowledge of the second language (e.g. sufficient vocabulary in the second language).
  3. Automaticity hypothesis/transfer facilitation model
    This states that for transfer of knowledge to occur, the word recognition (i.e. decoding skills) in the second language needs to be automatic past a threshold. For this to occur, the orthographies of the two languages need to be comparable. A good command of skills (e.g. reading strategies) in the first language is needed and this can be transferred if decoding is automatic in the second language.

In a structural model of languages, there are relations between skills in language one and language two. Lower

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Childhood: Clinical and School Psychology – Lecture 7 (UNIVERSITY OF AMSTERDAM)

Childhood: Clinical and School Psychology – Lecture 7 (UNIVERSITY OF AMSTERDAM)

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Specific learning disabilities are detrimental to one’s well-being. The repeated academic failure associated with this continues to haunt people for years after formal schooling.

A specific learning disability originally included a discrepancy-based definition. This included a discrepancy between IQ and achievement (e.g. high IQ, low reading achievement). However, the degree of discrepancy is arbitrary. This definition favours older children and children with a higher IQ. It is failure-based (e.g. waiting for children to show failure in academics).

The current definition is focused on identification with response to intervention (RTI). It includes achievement in key academic areas that is substantially below the age norm and in excess of sensory deficit (1), linguistic processes (2), attention (3) and memory (4). The prevalence rate is about 2% to 10%.

There are often social deficits in people with SLD but this may be because they get rejected and neglected by peers more often. People with SLD typically have low self-esteem and a poor self-esteem.

The DSM-5 definition of a specific learning disorder includes the following:

  • Difficulties in learning and using academic skills for at least six months.
  • The affected academic skills are substantially and quantifiably below those expected for the chronological age and lead to impairments in adaptive functioning.
  • The learning difficulties begin during school-age years but may not fully manifest themselves until the affected academic skills exceed the individual’s limited capacities (e.g. in the case of a timed test).
  • The learning difficulties are not better accounted for by other disorders (e.g. intellectual disability) or educational instruction.

The DSM-5 uses a discrepancy definition but not an IQ-discrepancy definition. There are three specifiers for severity:

  • Mild
    This includes some difficulties in learning in one or two academic domains but mild enough to still function well when provided with appropriate accommodations or support services.
  • Moderate
    This includes marked difficulties in learning skills in one or more academic domains. The individual is unlikely to become proficient without intensive or specialized training during the school years. Additional support may be needed.
  • Severe
    This includes severe difficulties in learning skills in several academic domains. The individual is unlikely to learn those skills without ongoing intensive, individualized and specialized training for most of the school years. Performance may be inadequate even with a lot of support.

Positive psychology interventions (PPI) aim to enhance well-being by increasing positive affect, cognition and behaviour (e.g. developing hope; mastery of life). This includes teaching coping skills. This approach holds that dyslexics have unique strengths and there needs to be a focus on this. However, not all dyslexics have unique strengths and their problems need to be addressed. Positive behavioural interventions (PBI) are more common and focus on providing remedies for the problematic behaviours associated with a specific learning disorder. Changing one’s mindset can help in alleviating the negative aspects of a specific

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